BL-RQ-001 · Open

Does restoring colonocyte butyrate oxidation lower luminal oxygen availability in humans, as it does in mouse models?

The mouse epithelial-oxygenation work is the mechanistic backbone of the ecology argument. Its human quantitative status is unknown.

Relation to the Host Capacity Model

HCM integrates the animal oxygen-gradient result into a human framework. That bridge is an inference, not evidence.

Measurements required to answer it

  • Mucosal oxygen tension or a validated surrogate
  • Faecal/mucosal butyrate with method reporting
  • Facultative anaerobe relative abundance

Confounders

  • Diet and fibre intake variation
  • Transit time
  • Sampling site (stool is not mucosa)
  • Antibiotic and PPI exposure

Decision criteria

A human study measuring mucosal oxygenation before and after a butyrate-restoring intervention with paired ecology data.

What would count against the Host Capacity Model

Human mucosal oxygenation shows no relationship with butyrate availability or facultative anaerobe expansion.

Registered claims this question bears on

  • BL-CLAIM-COL-002 · Animal evidence

    In mice, butyrate-driven PPAR-gamma signalling maintains epithelial oxygen consumption and keeps luminal oxygen tension low.

  • BL-CLAIM-COL-003 · HCM integration inference

    In a subset of people with persistent gut-centred chronic illness, impaired colonocyte butyrate oxidation and facultative-anaerobe expansion may form a self-reinforcing loop that resists single-target treatment.

Entities

  • ColonocyteThe absorptive epithelial cell of the colon. Its energy metabolism is unusual: it derives a large share of its ATP from beta-oxidation of the short-chain fatty acid butyrate rather than from glucose.
  • ButyrateA four-carbon short-chain fatty acid produced by bacterial fermentation of fibre in the colon and used as a primary respiratory fuel by colonocytes.
  • Butyrate oxidationMitochondrial beta-oxidation of butyrate inside colonocytes. Because this pathway consumes oxygen, its activity is one determinant of how much oxygen reaches the gut lumen.
  • Epithelial oxygenationThe oxygen gradient across the colonic mucosa. A steep gradient — low luminal oxygen — is a normal feature of a healthy colon and favours obligate anaerobes.

Cite this question

Attallah, M. BL-RQ-001: Does restoring colonocyte butyrate oxidation lower luminal oxygen availability in humans, as it does in mouse models? BiomeLogic research question registry. https://biomelogic.net/research-questions/BL-RQ-001

Machine-readable record (JSON)

Add BiomeLogic as a preferred source in Google

If you want to see this work more often in Google Search results, you can mark BiomeLogic as a preferred source. This is a Google setting you control; it does not change how any page here is ranked.

All open research questions