Why Dysbiosis, SIBO, Hydrogen Sulfide Intolerance, MCAS, and Long COVID May All Be Downstream of a Single Event — What this changes in practice

If the model is right, the entire sequence of care shifts. The standard approach asks, which organisms do I add or remove? The Host Capacity approach asks first, can the host actually sustain the ecosystem I'm trying to rebuild? — and if the answer is no, it works on capacity before, or alongside, working on the bugs. It means recognizing that pushing butyrate, fiber, NAC, glutathione, or probiotics into a host whose machinery is broken can be useless or actively counterproductive — and knowing which patient is which before you reach for the bottle. It means treating sulfur intolerance and glutathione depletion as capacity signals to be decoded, not deficiencies to be brute-forced. It means reading dysbiosis as a message about the terrain, not just a list of targets. None of this dismisses the microbiome. The microbes are real, they drive inflammation and metabolite imbalance and immune activation, and they absolutely belong in the analysis. But the host decides whether the ecosystem is allowed to recover. If the host cannot hold its oxygen gradient, run its mitochondria, recycle its glutathione, clear its sulfide, and keep its barrier intact, then no amount of seeding will make the garden grow.